Hypertension renal arteriolar sclerosis

　　Summary: department of primary hypertension caused by benign arteriolar nephrosclerosis (also known as hypertension renal arteriolar sclerosis) and malignant arteriolar nephrosclerosis, accompanied by corresponding clinical manifestations of the disease. Age more than in 40 ~ 50 years of age or older, hypertension 5 ~ 10 years of history, early only nocturia increased, then appear proteinuria, individual cases can occur due to the broken capillaries of transient with macroscopic haematuria, but without obvious lumbago, often merge arteriosclerosis sex retinopathy, left ventricular hypertrophy, coronary heart disease, heart failure, cerebral arteriosclerosis and (or) history of cerebrovascular accident.
　　Causes:
　　1.the weight and obese factors: weight gain is not only lead to obesity, is also a risk factor for hypertension, the more body fat, the greater the risk of hypertension.
　　2.high salt diet
　　3.drink
　　4.mental psychological factors: work tension, mental burden is heavy, irregular life, long-term stay up late, lack of sleep or rest, all can lead to high blood pressure
　　5.the body calcium is also one of the leading causes of high blood pressure disease happens.
　　6.genetic factors: study at home and abroad, high blood pressure have a strong genetic predisposition, but genetic factors and environmental factors interact with each other only can lead to a rise in blood pressure.
　　7.weather factors: because of the weather gets cold, the skin and subcutaneous tissue blood vessels to contract, causing peripheral vascular resistance increases, leading to increased blood pressure.
　　Features: slow progression of symptoms a few gradually develop into renal failure, most perennial mild renal function damage and abnormal urine routine, malignant high blood pressure, diastolic blood pressure should be more than 16 kpa (120 MMHG), accompanied by obvious cerebrovascular complications and rapid development, a large amount of proteinuria, often accompanied by hematuria, progressive renal function decline.
　　Early prevention:
　　first, the age of 40 and 50 years of age or older, hypertension 5-10 years of history, if determined to trace albumin increase, should be on high alert.
　　Second, nocturia increased, appear proteinuria or transient hematuria, will always check the renal function, urinary protein qualitative, quantitative, 24 hours urinary protein attention to measure blood pressure, do fundus examination.
　　Third, maintain defecate unobstructed.
　　Iv. Avoid contact with heavy metals, toxic substances and possible damage to the kidney drug.
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